*Note — My current focus is researching COVID-19’s origins, as part of the D.R.A.S.T.I.C. team of scientists, journalists & researchers. Recent news: D.R.A.S.T.I.C.’s research forms a large portion of the basis for investigations begun by the US Senate, House & National Institutes of Health. Recent appearances and/or discussion on 60 Minutes, The Joe Rogan Exp., Fox News, JRE [again], Bill Maher, & CNN. I am thankful for the efforts of @TheSeeker, @gilles@Harvard2H, @DaoYu15, among others, for placing the breadcrumbs that encouraged me to begin digging deeper into this set of anomalies.
I originally published this as a PDF on Google Docs in response to a ridiculous example of scientific smothering of actual debate. I learned a tough lesson - if you want to make fun of science by creating a fake journal and a fake article, published by a fake non-profit, you should at least advertise that it exists.
So, as I work to finish two works of actual research [after returning 18 hours ago from a family trip to Disney World [thanks grandma/grandpa!], here’s what I sent to Nature’s Amy Maxmen 6 weeks ago.
I’ve yet to receive a response…
I have some the interpretation of the "ACE2 and TMPRSS2 polymorphism analysis" paper referred to in your recent interview.
When checking the metadata and not being concerned about race specificity is looking at only immediate death. The paper refers to the polymorphism risk. The polymorphism in the paper is regarding the misfolding and wont show up for some time in the metadata. Background: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3904311/
This is the paper that was brought up today: https://bmcmedicine.biomedcentral.com/articles/10.1186/s12916-020-01673-z
Nobody seems to understand this paper correctly. Everyone seems to interpret in terms of acute effects. Incorrect!